Fluorinated topical steroids list

Doses of 125 to 250 mg/kg, administered intraperitoneally, have been shown to induce chromosomal aberrations and changes in chromosome organization of spermatogonia in rats. Spermatogonial differentiation was also inhibited by fluorouracil, resulting in transient infertility. However, in studies with a strain of mouse which is sensitive to the induction of sperm head abnormalities after exposure to a range of chemical mutagens and carcinogens, fluorouracil was inactive at oral doses of 5 to 80 mg/kg/day. In female rats, fluorouracil administered intraperitoneally at doses of 25 and 50 mg/kg during the preovulatory phase of oogenesis significantly reduced the incidence of fertile matings, delayed the development of preimplantation and postimplantation embryos, increased the incidence of preimplantation lethality and induced chromosomal anomalies in these embryos. Single dose intravenous and intraperitoneal injections of 5-fluorouracil have been reported to kill differentiated spermatogonia and spermatocytes (at 500 mg/kg) and to produce abnormalities in spermatids (at 50 mg/kg) in mice.

The obvious priority is immediate discontinuation of any further topical corticosteroid use. Protection and support of the impaired skin barrier is another priority. Eliminating harsh skin regimens or products will be necessary to minimize potential for further purpura or trauma, skin sensitivity, and potential infection. Steroid Atrophy [10] [11] is often permanent, though if caught soon enough and the topical corticosteroid discontinued in time, the degree of damage may be arrested or slightly improve. However, while the accompanying Telangectasias may improve marginally, the Striae is permanent and irreversible. [1]

Vehicle

  1. If lesion is dry - use ointment or moisturizing lotion
  2. If moist or weeping - use cream (or gel)
  3. If in hair covered area - consider lotion or liquid preparations
Strength (based on thickness of skin and severity and thickness of lesion)
  1. Face and genitals - weakest strength - thin skin, avoid atrophy
  2. Hands and feet - thickest skin - if lesion is severe and thickened (lichenified) may need most potent strength
  3. Other areas - use strength appropriate to severity and thickness of lesions
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Penile carcinoma in situ is a premalignant lesion restricted to the skin. It typically affects uncircumcised men older than 60 years. Velvety plaques of the glans penis are known as erythroplasia of Queyrat. Keratotic plaques are known as Bowen disease ( Figure 3A ) , which occurs on the penile shaft, scrotal skin, or perineum. 6 , 19 Human papilloma virus (HPV) is the primary etiology of penile carcinoma in situ, although other factors may include smegma and trauma from friction, heat, and inflammation. 40 Penile carcinoma in situ progresses to squamous cell carcinoma in approximately 5 to 30 percent of patients. 41 , 42

Fluorinated topical steroids list

fluorinated topical steroids list

Penile carcinoma in situ is a premalignant lesion restricted to the skin. It typically affects uncircumcised men older than 60 years. Velvety plaques of the glans penis are known as erythroplasia of Queyrat. Keratotic plaques are known as Bowen disease ( Figure 3A ) , which occurs on the penile shaft, scrotal skin, or perineum. 6 , 19 Human papilloma virus (HPV) is the primary etiology of penile carcinoma in situ, although other factors may include smegma and trauma from friction, heat, and inflammation. 40 Penile carcinoma in situ progresses to squamous cell carcinoma in approximately 5 to 30 percent of patients. 41 , 42

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